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Tuberculosis is a chronic infectious granulomatous disease
caused by Mycobacterium tuberculosis
1 which is an aerobic,
slender, non-motile, non-encapsulated, non-sporing, rod
shaped organism ranging from 2 to 5 µm2
. The World
Health Organization (WHO) estimates that worldwide
there are approximately 20 million active cases, of them
approximately 3 million people die each year from
tuberculosis, of which 80 % are in developing countries
3
.
Tuberculous oral lesions are relatively rare occurrence.
Oral manifestations occur in approximately 3% of cases
involving long standing pulmonary and/ or systemic
infection4
.Oral clinical presentation may be as ulcers,
erythematous patches, and indurated lesions with granular
surface, nodules, and fissures or as jaw lesions. The most
common sites involved are tongue, gingiva, tooth sockets
and jaw involvement may present as osteomyelitis
5
.
Two main types of tubercular infections of oral
tissues are recognized – Primary and Secondary. Primary
lesions develop when tuberculosis bacilli are directly
inoculated into the oral tissues of a person who has not
acquired immunity to the disease and in fact, any area that
is vulnerable to direct inoculation of bacilli from
exogenous source can be a potential site. These frequently
involve gingiva, tooth extraction sockets and buccal folds.
Secondary infection of oral tissues can result from either
haematogenous or lymphatic spread or from
autoinoculation by infected sputum and direct extensions
from neighbouring structures. Intraoral sites frequentlyinvolved include the tongue, palate, lips, alveolar mucosa
and jaw bones
2
.
With myriad presentations and sometimes lack of
specific systemic symptoms, oral tubercular lesions may
present as puzzle for us and may escape our eyes. Hence,
we document a case of primary tuberculous osteomyelitis
of mandible in an old male individual who was initially
suspected for dental abscess with nonspecific chronic
osteomyelitis and later proved as primary tubercular osteomylitis